OncoMed Publishes Data on Tarextumab’s Anti-Cancer Stem Cell Activity in Clinical Cancer Research

On May 1, 2015 OncoMed Pharmaceuticals reported that data summarizing preclinical study results for its anti-Notch2/3 antibody, tarextumab (OMP-59R5) were published in the May 1, 2015 edition of Clinical Cancer Research (Press release, OncoMed, MAY 1, 2015, View Source [SID:1234503253]). The article details tarextumab’s anti-tumor effects alone and in combination with chemotherapy and elucidates the drug candidate’s mechanisms of activity. Additionally, data is presented describing the basis for biomarker identification of tumors with increased sensitivity to treatment with tarextumab.

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"The early evidence of activity that we are seeing for tarextumab in our clinical trials is consistent with results of our preclinical studies. For instance, the Phase 1b biomarker-delineated response rates and survival data for tarextumab in pancreatic cancer is similar to the preclinical evidence that tumors with high Notch3 gene expression are more likely to respond to treatment," said Paul J. Hastings, OncoMed’s Chairman and Chief Executive Officer. "Tarextumab is currently being studied in two randomized Phase 2 clinical trials for pancreatic cancer and small cell lung cancer. We look forward to reporting additional clinical and biomarker data from our tarextumab program in the months to come."

Key findings from the paper, titled "Targeting Notch signaling with a Notch2/Notch3 antagonist (tarextumab) inhibits tumor growth and decreases tumor initiating cell frequency", include:

Tarextumab was efficacious in inhibiting the growth of preclinical patient-derived xenografts from various solid tumor types, including lung, ovarian, breast and pancreatic cancers, indicating the potential for broad utility of tarextumab.

Tarextumab demonstrated a dual mechanism of action: inhibiting signaling of Notch2 and Notch3 in tumor cells and modulating the function of tumor vasculature through its action on pericytes.

Inhibition of Notch signaling in tumor cells was associated with a reduction in cancer stem cell frequency, promotion of tumor cell differentiation and a delay in tumor recurrence following chemotherapy treatment.

Tumor sensitivity to treatment with tarextumab in combination with chemotherapy was significantly higher in pancreatic tumors that had a higher level of Notch3 gene expression.

Based on these data, Notch3 gene expression is being evaluated as a potential predictive biomarker for tarextumab in ongoing clinical trials.

"In this paper, we show that the blockade of Notch signaling with tarextumab sensitizes tumors to chemotherapy and reduces cancer stem frequency, delaying cancer recurrence as compared to chemotherapy alone," said Tim Hoey, Ph.D., Senior Vice President, Cancer Biology and a co-author of the paper. "Preclinical data for tarextumab, particularly as part of a combination regimen, provide a strong rationale for the utility of targeting Notch2 and Notch3 for cancer treatment, and suggest that this therapeutic approach may improve clinical outcomes. We are currently exploring the impact of this targeted anti-cancer stem cell agent in our Phase 2 clinical trials."

About Tarextumab (anti-Notch2/3, OMP-59R5)

Tarextumab (anti-Notch2/3, OMP-59R5) is a fully human monoclonal antibody that targets the Notch2 and Notch3 receptors. Preclinical studies have suggested that tarextumab exhibits two mechanisms of action: (1) by downregulating Notch pathway signaling, tarextumab appears to have anti-cancer stem cell effects, and (2) tarextumab affects pericytes, impacting stromal and tumor microenvironment. Tarextumab is currently being studied in two randomized Phase 2 clinical trials. The "ALPINE" study is assessing tarextumab with Abraxane (paclitaxel protein-bound particles for injectable suspension) (albumin bound) plus gemcitabine in first-line advanced pancreatic cancer patients. The "PINNACLE" study is testing tarextumab in combination with etoposide and cisplatin and etoposide and carboplatin in first-line extensive stage small cell lung cancer patients. Data from OncoMed’s Phase 1a and Phase 1b clinical trials of tarextumab indicate that the antibody is well tolerated, with on-target modulation of the Notch signaling pathway and signs of anti-tumor activity. Tarextumab is part of OncoMed’s collaboration with GlaxoSmithKline (GSK). GSK has an option to obtain an exclusive license to tarextumab during certain time periods through completion of the proof-of-concept Phase 2 trials.

10-Q – Quarterly report [Sections 13 or 15(d)]

Threshold Pharmaceuticals has filed a 10-Q – Quarterly report [Sections 13 or 15(d)] with the U.S. Securities and Exchange Commission (Filing, 10-Q, Threshold Pharmaceuticals, APR 30, 2015, View Source [SID1234503259]).

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Gilead Sciences Announces First Quarter 2015 Financial Results

On April 30, 2015 Gilead Sciences, Inc. (Nasdaq: GILD) reported its results of operations for the first quarter ended March 31, 2015 (Filing, Q1, Gilead Sciences, APR 30, 2015, View Source [SID:1234506608]). The financial results that follow represent a year over year comparison of first quarter 2015 to the first quarter 2014. Total revenues were $7.6 billion in 2015 compared to $5.0 billion in 2014. Product sales were $7.4 billion in 2015 compared to $4.9 billion in 2014. Net income was $4.3 billion, or $2.76 per diluted share in 2015 compared to $2.2 billion or $1.33 per diluted share in 2014. Non-GAAP net income, which excludes amounts related to acquisition, restructuring, stock-based compensation and other, was $4.6 billion, or $2.94 per diluted share in 2015 compared to $2.5 billion or $1.48 per diluted share in 2014.

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Product Sales

Total product sales for the first quarter of 2015 were $7.4 billion compared to $4.9 billion for the first quarter of 2014. In the first quarter, product sales in the U.S. were $5.2 billion compared to $3.6 billion for the first quarter of 2014, and in Europe, product sales for the first quarter of 2015 were $1.8 billion compared to $1.0 billion for the same period in 2014.

Antiviral Product Sales

Antiviral product sales increased to $7.0 billion for the first quarter of 2015, up from $4.5 billion for the first quarter of 2014 primarily due to sales of Harvoni (ledipasvir 90 mg/sofosbuvir 400 mg), which was approved in the U.S. and Europe in the fourth quarter of 2014, partially offset by a decrease in sales of Sovaldi (sofosbuvir).

Other Product Sales

Other product sales, which include Letairis, Ranexa and AmBisome, were $417 million for the first quarter of 2015 compared to $362 million for the first quarter of 2014.

Operating Expenses

During the first quarter of 2015, compared to the same period in 2014:

Non-GAAP research and development (R&D) expenses increased primarily due to the continued progression and expansion of Gilead’s clinical studies, particularly phase 3 studies in the liver disease and oncology areas.
Non-GAAP selling, general and administrative (SG&A) expenses increased primarily due to growth in our business including commercial expansion for our hepatitis C virus (HCV) products.

As of March 31, 2015, Gilead had $14.5 billion of cash, cash equivalents and marketable securities compared to $11.7 billion as of December 31, 2014. During the first quarter of 2015, Gilead generated $5.7 billion in operating cash flow and utilized $3.0 billion to repurchase shares, which completes our May 2014 stock repurchase program.

Revised 2015 Full Year Guidance

Gilead updated its full year 2015 guidance, which it initially provided on February 3, 2015.

Product & Pipeline Updates Announced by Gilead During the First Quarter of 2015 Include:

Antiviral Program

Announced that the Japanese Ministry of Health, Labour and Welfare approved Sovaldi for the suppression of viremia in patients with genotype 2 chronic HCV infection with or without compensated cirrhosis. Sovaldi is indicated for use in combination with ribavirin (RBV) for 12 weeks. Sovaldi (in combination with RBV) is the first all-oral, interferon-free treatment regimen for genotype 2 HCV infection.

Presentation of data at the 22nd Conference on Retroviruses and Opportunistic Infections included announcement of:
Positive results from a Phase 3 clinical trial evaluating the once-daily single tablet regimen Harvoni for the treatment of genotypes 1 or 4 chronic HCV infection among patients co-infected with HIV. In the trial, 96 percent of HCV patients achieved a sustained virologic response 12 weeks after completing therapy (SVR12). Patients who achieve SVR12 are considered cured of HCV infection.

Positive 48-week results from two Phase 3 studies (Studies 104 and 111) evaluating an investigational once-daily single tablet regimen containing tenofovir alafenamide (TAF) for the treatment of HIV-1 infection in treatment-naïve adults. TAF is a novel nucleotide reverse transcriptase inhibitor that has demonstrated high antiviral efficacy at a dose 10 times lower than Gilead’s Viread, as well as improved renal and bone laboratory parameters, in clinical trials.

Positive results from a preclinical study conducted in collaboration with researchers at Beth Israel Deaconess Medical Center evaluating a proprietary investigational oral TLR7 agonist and analogue of GS-9620 as part of an HIV eradication strategy. Data demonstrated that treatment with the TLR7 agonist induced transient plasma Simian Immunodeficiency Virus (SIV) RNA, as well as reduced SIV DNA in virally suppressed rhesus macaques given antiretroviral therapy (ART). In addition, the study found that after discontinuation of ART, SIV viral loads were lower among macaques that received the proprietary TLR7 agonist compared to the placebo group.

– See more at: View Source#sthash.uEsnwxvq.dpuf

10-Q – Quarterly report [Sections 13 or 15(d)]

Eli Lilly has filed a 10-Q – Quarterly report [Sections 13 or 15(d)] with the U.S. Securities and Exchange Commission (Filing, 10-Q, Eli Lilly, APR 30, 2015, View Source [SID1234503232]).

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10-Q – Quarterly report [Sections 13 or 15(d)]

Celgene has filed a 10-Q – Quarterly report [Sections 13 or 15(d)] with the U.S. Securities and Exchange Commission (Filing, 10-Q, Celgene, APR 30, 2015, View Source [SID1234503240]).

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