Natera to Present New Gastrointestinal Cancer Data at the ESMO Annual Meeting

On September 18, 2020 Natera, Inc. (NASDAQ: NTRA), a pioneer and global leader in cell-free DNA testing, reported it will present new data on its personalized molecular residual disease (MRD) test, Signatera, at the 2020 European Society for Medical Oncology (ESMO) (Free ESMO Whitepaper) virtual meeting taking place September 19-21, 2020 (Press release, Natera, SEP 18, 2020, View Source [SID1234565366]).

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Natera will present four abstracts — one oral and three poster presentations — which highlight applications for detecting MRD levels and tracking tumor clonal evolution in CRC and in esophageal adenocarcinoma.

"When CRC patients present with multiple lesions, recurrence monitoring may be complicated by spatial heterogeneity between the primary tumor and the metastases," said Andrés Cervantes, M.D., Ph.D., President-Elect of ESMO (Free ESMO Whitepaper) and co-author of the tumor evolution study. "Our study leverages a tumor-informed ctDNA technology that can evaluate and account for this heterogeneity."

"We’re delighted to share new data at this year’s ESMO (Free ESMO Whitepaper) meeting, which shows the broad potential of our tumor-informed approach to MRD assessment," said Alexey Aleshin, M.D., M.B.A., Senior Medical Director of Oncology at Natera. "These studies demonstrate that Signatera provides clinically actionable information that can improve the management of patients with multiple different solid cancers."

Details about the abstracts are as follows:

Abstract #405MO | Mini Oral Presentation
Presenter: Fotios Loupakis, M.D., Ph.D.

Personalized circulating tumor DNA assay for the detection of minimal residual disease in CRC patients undergoing resections of metastases.

This study evaluated the prognostic value of MRD testing after surgical resection of metastases. MRD positivity correlated with inferior progression-free survival and was the most significant biomarker for prognosis in patients with oligometastatic CRC.

Abstract #420P | Poster Presentation
Presenter: Tenna V. Henriksen, M.S.

MRD detection and tracking tumor evolution using ctDNA in stage I-III colorectal cancer patients

A prospective, multicenter cohort study that analyzed the genetic heterogeneity between primary tumor, metastases, and synchronous tumors in patients with stage I-III CRC using Signatera. While primary and metastatic tumors showed at least 50 percent phylogenetic similarity, synchronous CRC tumors were observed to be genetically unrelated and required individual Signatera assays to be designed for reliable ctDNA detection.

Abstract #1491P | Poster Presentation
Presenter: Emma Ococks, M.S.

Bespoke circulating tumor DNA assay for the detection of minimal residual disease in esophageal adenocarcinoma patients

A pilot study that evaluated the ability of tumor-informed ctDNA testing to assess MRD status in patients with esophageal adenocarcinoma, a low-shedding tumor type. Postoperative assessment of MRD detected clinical relapse with a median lead time of 335 days and with 100 percent sensitivity.

Abstract #520TiP | Poster Presentation
Presenter: Pashtoon Kasi, M.D., M.S.

A multicenter study to evaluate the impact of circulating tumor DNA guided therapy (BESPOKE) in patients with Stage II and III colorectal cancer

A trial-in-progress poster presentation, describing the first real-world, prospective, multicenter study (BESPOKE) utilizing a tumor-informed ctDNA test to assess MRD status in 1000 patients with stage II and III CRC over the course of two years. The primary endpoint of this study is to examine the impact of Signatera on adjuvant treatment decisions and to determine the rates of CRC recurrence while asymptomatic.

About Signatera
Signatera is a custom-built circulating tumor DNA (ctDNA) test for treatment monitoring and molecular residual disease (MRD) assessment in patients previously diagnosed with cancer. The test is available for clinical and research use, and in 2019, it was granted Breakthrough Device Designation by the FDA. The Signatera test is personalized and tumor-informed, providing each individual with a customized blood test tailored to fit the unique signature of clonal mutations found in that individual’s tumor. This maximizes accuracy for detecting the presence or absence of residual disease in a blood sample, even at levels down to a single tumor molecule in a tube of blood. Unlike a standard liquid biopsy, Signatera is not intended to match patients with any particular therapy; rather, it is intended to detect and quantify how much cancer is left in the body, to detect recurrence earlier and to help optimize treatment decisions. Signatera test performance has been clinically validated in multiple cancer types including colorectal, non-small cell lung, breast, and bladder cancers. Signatera has been developed and its performance characteristics determined by Natera, the CLIA-certified laboratory performing the test. The test has not been cleared or approved by the US Food and Drug Administration (FDA). CAP accredited, ISO 13485 certified, and CLIA certified.

BERG To Present Discovery/Validation Of Biomarkers Associated With Survival In Pancreatic Ductal Adenocarcinoma (PDAC) Treated With BPM 31510-IV At The European Society For Medical Oncology (ESMO) 2020 Congress

On September 18, 2020 BERG, a clinical-stage biotech that employs artificial intelligence (AI) to investigate diseases and develop innovative treatments, reported two major medical/clinical research developments on pancreatic ductal adenocarcinoma (PDAC) to be presented virtually at the European Society for Medical Oncology (ESMO) (Free ESMO Whitepaper) 2020 Congress taking place from September 19-21, 2020 (Press release, Berg, SEP 18, 2020, View Source [SID1234565365]).

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The first study entitled "Project Survival: High Fidelity Longitudinal Phenotypic and Multi-omic Characterization of Pancreatic Ductal Adenocarcinoma (PDAC) for Biomarker Discovery", is the culmination of the largest existing high-fidelity characterization of pancreatic cancer from a phenotypic/adaptive multi-omic perspective. BERG’s Interrogative Biology platform was employed to identify causal relationships between existing pancreatic cancer therapies and changes in proteomic, metabolic and lipidomic responses to 253 treatment interventions and 211 progression events.

The research cohort included PDAC patients across different stages including early, locally advanced and metastatic to yield the most accurate characterization of the evolution of the disease. Throughout the course of the study, 470,000 clinical data points were gathered.

"Valid biomarkers will have a dramatically positive impact on patients suffering from pancreatic cancer," said Dr. A. James Moser, Co-Director of the Pancreas and Liver Institute at Beth Israel Deaconess Medical Center (BIDMC), and Professor of Surgery at Harvard Medical School. "The Project Survival and BPM study data cross an important threshold on the road to personalized treatment, underscoring the vital role of comprehensive and sustained research collaborations in the fight against this dreadful cancer."

The second study, Validation of Response and Survival Biomarkers in a Phase 2 Trial of BPM 31510—IV Advanced Refractory Pancreatic Ductal Adenocarcinoma (PDAC), identified two potential biomarkers predictive of response and overall survival in PDAC patients in response to BPM 31510-IV treatment.

The Interrogative Biology platform was applied to patient Buffy Coat (BC) samples being treated with BERG’s pancreatic drug cancer candidate, BPM 31510-IV, in a Phase 1 trial to identify novel markers. The biomarkers were then independently confirmed for utility between survival (OS), stable disease (SD), and progressive disease (PD) in a Phase 2 trial.

"PDAC is the most common type of pancreatic cancer and is an infamously devastating disease," said Dr. Niven R. Narain, BERG Co-founder, President and Chief Executive Officer. "We are fully committed to continue advancing these critical developments and leveraging our proprietary Interrogative Biology platform to identify therapeutic improvements for patients. We’re honored to present these findings at the ESMO (Free ESMO Whitepaper) 2020 Congress, and continue fostering partnerships with pioneers across the oncology field."

Titan Pharmaceuticals’ Stockholders Approve Proposal To Amend Its Certificate Of Incorporation

On September 18, 2020 Titan Pharmaceuticals, Inc. (NASDAQ: TTNP) ("Titan" or the "Company") reported that, at today’s special meeting, its stockholders approved the proposal to amend Titan’s certificate of incorporation to increase the number of authorized shares of common stock from 125 million to 225 million (the "Amendment") (Press release, Titan Pharmaceuticals, SEP 18, 2020, View Source [SID1234565364]).

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The voting results are available in our Current Report on Form 8-K filed with the SEC today.

"On behalf of the Board and management team, I would like to thank our stockholders for their continued support and we look forward to updating you on our progress," said Titan’s Executive Chairman, Dr. Marc Rubin.

Takeda Presents New Data Highlighting Scientific Advancements in Lung Cancer at ESMO Virtual Congress

On September 18, 2020 Takeda Pharmaceutical Company Limited (TSE:4502/NYSE:TAK) ("Takeda") reported that the company is presenting data from its lung cancer portfolio at the virtual European Society for Medical Oncology (ESMO) (Free ESMO Whitepaper) conference (Press release, Takeda, SEP 18, 2020, View Source [SID1234565363]). Notably, insights from sub-analyses of the Phase 3 ALTA 1L study reinforce both the compelling evidence of intracranial efficacy with ALUNBRIG (brigatinib) as a first-line treatment for patients with anaplastic lymphoma kinase-positive (ALK+) non-small cell lung cancer (NSCLC) as well as associated quality of life (QoL) data. Takeda is also featuring updated 10-month follow-up results from the Phase 1/2 trial of mobocertinib (TAK-788), demonstrating mobocertinib achieved a duration of response (DoR) of more than one year in the trial’s study population of patients with epidermal growth factor receptor (EGFR) Exon20 insertion+ metastatic NSCLC (mNSCLC).

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"We’re pleased to present our ongoing research in lung cancer at this year’s virtual ESMO (Free ESMO Whitepaper) congress, including new results from our ongoing Phase 3 ALTA 1L trial, which reinforce ALUNBRIG’s superiority over crizotinib and underscore the strength of the data upon which ALUNBRIG earned its recent first-line indication in the U.S. and European Union," said Christopher Arendt, Head, Oncology Therapeutic Area Unit, Takeda. "Additionally, updated data from the Phase 1/2 trial of mobocertinib showed a prolonged duration of response in patients with EGFR Exon20 insertion+ mNSCLC, representing promising progress for an underserved population that derives limited efficacy from currently available treatments."

Results from sub-analyses of the Phase 3 ALTA 1L study, featured in two poster sessions, provide additional insight into ALUNBRIG’s intracranial efficacy and improvements in QoL in the first-line setting. ALUNBRIG significantly prolonged time to intracranial disease progression versus crizotinib in patients with ALK tyrosine kinase inhibitor (TKI)-naïve ALK+ NSCLC, showcasing robust intracranial activity. Key findings as assessed by a blinded independent review committee (BIRC) among patients with baseline brain metastases showed:

An average of 24 months (12.9–NR) passed before intracranial disease progression in patients treated with ALUNBRIG compared to 5.6 months for crizotinib (4.0–9.2).
ALUNBRIG demonstrated a significant improvement in overall progression-free survival (PFS) in patients who had no prior brain radiotherapy, yielding an average of 24 months versus 5.5 months with crizotinib (hazard ratio [HR]: 0.27, P=0.0003).
The brain was the first site of disease progression less frequently in patients treated with ALUNBRIG (31%) than with crizotinib (42%).
ALUNBRIG continued to demonstrate improvements in global health status (GHS)/QoLcompared to crizotinib with significantly delayed time to deterioration in patients with baseline brain metastases (16.6 months versus 4.7 months for crizotinib [HR: 0.54, 95% CI 0.29‒1.00; P=0.0415]).
Serious adverse reactions occurred in 33% of patients receiving ALUNBRIG. The most common serious adverse reactions other than disease progression were pneumonia (4.4%), ILD/pneumonitis (3.7%), pyrexia (2.9%), dyspnea (2.2%), pulmonary embolism (2.2%) and asthenia (2.2%). Fatal adverse reactions other than disease progression occurred in 2.9% of patients and included pneumonia (1.5%), cerebrovascular accident (0.7%) and multiple organ dysfunction syndrome (0.7%).
"Ongoing results from the ALTA 1L trial demonstrate brigatinib as an effective first-line treatment option for ALK+ NSCLC patients, especially those with baseline brain metastases," said Professor Sanjay Popat, Consultant Medical Oncologist, The Royal Marsden NHS Foundation Trust. "While we now have several targeted treatment options available to treat the disease, therapies that have demonstrated robust intracranial efficacy have shown to be an invaluable addition as we seek to extend and improve the lives of our patients."

Key findings from an on-demand mini oral session featuring results from the Phase 1/2 trial of mobocertinib with 10 months of follow up will also be presented. Confirmed responders treated with mobocertinib maintained a response for an average of more than one year, with a median DoR of 13.9 months (5.0-NR). With additional follow-up, the trial maintained a 43% (12/28; 95% CI 24‒63) confirmed objective response rate (ORR) and a median PFS of 7.3 months (95% CI 4.4‒15.6) among the total patients treated. The safety profile of mobocertinib was manageable. For patients with EGFR Exon20 insertion+ mNSCLC treated at the 160 mg once daily dose, the most common adverse reactions (>=25%) were diarrhea (89%), decreased appetite (54%), vomiting (54%), rash (46%), nausea (46%) and anemia (36%). Among all patients treated at the 160 mg once daily dose, the most common treatment-related adverse events (TRAEs) were diarrhea (88%), nausea (49%), vomiting (36%) and rash (35%).

Data from the pivotal Phase 2 extension cohort of mobocertinib, EXCLAIM, will read out in fiscal year 2020.

About ALUNBRIG (brigatinib)

ALUNBRIG is a potent and selective next-generation tyrosine kinase inhibitor (TKI) that was designed to target anaplastic lymphoma kinase (ALK) molecular alterations.

ALUNBRIG is approved in the U.S. and European Union (EU) as a first-line treatment for patients with ALK-positive (ALK+) metastatic non-small cell lung cancer (mNSCLC) previously not treated with an ALK inhibitor. ALUNBRIG is also approved in more than 40 countries, including the U.S., Canada and the EU, for the treatment of people living with ALK+ mNSCLC who have taken the medicine crizotinib, but their NSCLC has worsened or they cannot tolerate taking crizotinib.

About ALK+ NSCLC

Non-small cell lung cancer (NSCLC) is the most common form of lung cancer, accounting for approximately 85% of the estimated 1.8 million new cases of lung cancer diagnosed each year worldwide, according to the World Health Organization.1,2 Genetic studies indicate that chromosomal rearrangements in anaplastic lymphoma kinase (ALK) are key drivers in a subset of NSCLC patients.3 Approximately three to five percent of patients with metastatic NSCLC have a rearrangement in the ALK gene.4,5,6 ALK+ NSCLC is a complex and nuanced disease that presents treatment challenges for newly diagnosed patients, especially those whose disease has spread to their brain.

Takeda is committed to continuing research and development in NSCLC to improve the lives of the approximately 40,000 patients diagnosed with this serious and rare form of lung cancer worldwide each year.7

About Mobocertinib (TAK-788)

Mobocertinib is a potent, small-molecule tyrosine kinase inhibitor (TKI) specifically designed to selectively target epidermal growth factor receptor (EGFR) and human EGFR2 (HER2) Exon20 insertion mutations. In 2019, the U.S. FDA granted mobocertinib Orphan Drug Designation for the treatment of lung cancer with HER2 mutations or EGFR mutations including Exon20 insertion mutations. In April 2020, mobocertinib received Breakthrough Therapy Designation from the FDA for patients with EGFR Exon20 insertion+ metastatic non-small cell lung cancer (mNSCLC) whose disease has progressed on or after platinum-based chemotherapy.

The mobocertinib development program began in the NSCLC population and is expected to expand to additional underserved populations in other tumor types. Mobocertinib is an investigational drug for which efficacy and safety have not been established.

About EGFR Exon20 Insertion+ mNSCLC

Patients with epidermal growth factor receptor (EGFR) Exon20 insertion+ metastatic non-small cell lung cancer (mNSCLC) make up only about 1-2% of patients with NSCLC.8,9 This disease carries a worse prognosis than other EGFR mutations because there are currently no FDA-approved therapies that target Exon20 mutations, and current EGFR TKIs and chemotherapy provide limited benefit for these patients.

Takeda is committed to continuing research and development in EGFR Exon20 insertion+ mNSCLC with the hope of introducing a targeted treatment option for the approximately 30,000 patients diagnosed with the disease worldwide each year.8,9

ALUNBRIG IMPORTANT SAFETY INFORMATION

WARNINGS AND PRECAUTIONS

Interstitial Lung Disease (ILD)/Pneumonitis: Severe, life-threatening, and fatal pulmonary adverse reactions consistent with interstitial lung disease (ILD)/pneumonitis have occurred with ALUNBRIG. In the ALUNBRIG arm of trial ALTA 1L (180 mg once daily), ILD/pneumonitis occurred in 5.1% of patients receiving ALUNBRIG. ILD/pneumonitis occurred within 8 days of initiation of ALUNBRIG in 2.9% of patients, with Grade 3 to 4 reactions occurring in 2.2% of patients. In Trial ALTA, ILD/pneumonitis occurred in 3.7% of patients in the 90 mg group (90 mg once daily) and 9.1% of patients in the 90→180 mg group (180 mg once daily with 7-day lead-in at 90 mg once daily). Adverse reactions consistent with possible ILD/pneumonitis occurred early within 9 days of initiation of ALUNBRIG (median onset was 2 days) in 6.4% of patients, with Grade 3 to 4 reactions occurring in 2.7%. Monitor for new or worsening respiratory symptoms (e.g., dyspnea, cough, etc.), particularly during the first week of initiating ALUNBRIG. Withhold ALUNBRIG in any patient with new or worsening respiratory symptoms, and promptly evaluate for ILD/pneumonitis or other causes of respiratory symptoms (e.g., pulmonary embolism, tumor progression, and infectious pneumonia). For Grade 1 or 2 ILD/pneumonitis, either resume ALUNBRIG with dose reduction after recovery to baseline or permanently discontinue ALUNBRIG. Permanently discontinue ALUNBRIG for Grade 3 or 4 ILD/pneumonitis or recurrence of Grade 1 or 2 ILD/pneumonitis.

Hypertension: In the ALUNBRIG arm of trial ALTA 1L (180 mg once daily), hypertension was reported in 32% of patients receiving ALUNBRIG; Grade 3 hypertension occurred in 13% of patients. In ALTA, hypertension was reported in 11% of patients in the 90 mg group who received ALUNBRIG and 21% of patients in the 90→180 mg group. Grade 3 hypertension occurred in 5.9% of patients overall. Control blood pressure prior to treatment with ALUNBRIG. Monitor blood pressure after 2 weeks and at least monthly thereafter during treatment with ALUNBRIG. Withhold ALUNBRIG for Grade 3 hypertension despite optimal antihypertensive therapy. Upon resolution or improvement to Grade 1, resume ALUNBRIG at the same dose. Consider permanent discontinuation of treatment with ALUNBRIG for Grade 4 hypertension or recurrence of Grade 3 hypertension. Use caution when administering ALUNBRIG in combination with antihypertensive agents that cause bradycardia.

Bradycardia: In the ALUNBRIG arm of trial ALTA 1L (180 mg once daily), heart rates less than 50 beats per minute (bpm) occurred in 8.1% of patients receiving ALUNBRIG. Grade 3 bradycardia occurred in 1 patient (0.7%). In ALTA, heart rates less than 50 beats per minute (bpm) occurred in 5.7% of patients in the 90 mg group and 7.6% of patients in the 90→180 mg group. Grade 2 bradycardia occurred in 1 (0.9%) patient in the 90 mg group. Monitor heart rate and blood pressure during treatment with ALUNBRIG. Monitor patients more frequently if concomitant use of drug known to cause bradycardia cannot be avoided. For symptomatic bradycardia, withhold ALUNBRIG and review concomitant medications for those known to cause bradycardia. If a concomitant medication known to cause bradycardia is identified and discontinued or dose adjusted, resume ALUNBRIG at the same dose following resolution of symptomatic bradycardia; otherwise, reduce the dose of ALUNBRIG following resolution of symptomatic bradycardia. Discontinue ALUNBRIG for life-threatening bradycardia if no contributing concomitant medication is identified.

Visual Disturbance: In the ALUNBRIG arm of trial ALTA 1L (180 mg once daily), Grade 1 or 2 adverse reactions leading to visual disturbance including blurred vision, photophobia, photopsia, and reduced visual acuity were reported in 7.4% of patients receiving ALUNBRIG. In ALTA, adverse reactions leading to visual disturbance including blurred vision, diplopia, and reduced visual acuity, were reported in 7.3% of patients treated with ALUNBRIG in the 90 mg group and 10% of patients in the 90→180 mg group. Grade 3 macular edema and cataract occurred in one patient each in the 90→180 mg group. Advise patients to report any visual symptoms. Withhold ALUNBRIG and obtain an ophthalmologic evaluation in patients with new or worsening visual symptoms of Grade 2 or greater severity. Upon recovery of Grade 2 or Grade 3 visual disturbances to Grade 1 severity or baseline, resume ALUNBRIG at a reduced dose. Permanently discontinue treatment with ALUNBRIG for Grade 4 visual disturbances.

Creatine Phosphokinase (CPK) Elevation: In the ALUNBRIG arm of trial ALTA 1L (180 mg once daily), creatine phosphokinase (CPK) elevation occurred in 81% of patients who received ALUNBRIG. The incidence of Grade 3 or 4 CPK elevation was 24%. Dose reduction for CPK elevation occurred in 15% of patients. In ALTA, CPK elevation occurred in 27% of patients receiving ALUNBRIG in the 90 mg group and 48% of patients in the 90 mg→180 mg group. The incidence of Grade 3‑4 CPK elevation was 2.8% in the 90 mg group and 12% in the 90→180 mg group. Dose reduction for CPK elevation occurred in 1.8% of patients in the 90 mg group and 4.5% in the 90→180 mg group. Advise patients to report any unexplained muscle pain, tenderness, or weakness. Monitor CPK levels during ALUNBRIG treatment. Withhold ALUNBRIG for Grade 3 or 4 CPK elevation with Grade 2 or higher muscle pain or weakness. Upon resolution or recovery to Grade 1 CPK elevation or baseline, resume ALUNBRIG at the same dose or at a reduced dose.

Pancreatic Enzyme Elevation: In the ALUNBRIG arm of trial ALTA 1L (180 mg once daily), amylase elevation occurred in 52% of patients and Grade 3 or 4 amylase elevation occurred in 6.8% of patients. Lipase elevations occurred in 59% of patients and Grade 3 or 4 lipase elevation occurred in 17% of patients. In ALTA, amylase elevation occurred in 27% of patients in the 90 mg group and 39% of patients in the 90→180 mg group. Lipase elevations occurred in 21% of patients in the 90 mg group and 45% of patients in the 90→180 mg group. Grade 3 or 4 amylase elevation occurred in 3.7% of patients in the 90 mg group and 2.7% of patients in the 90→180 mg group. Grade 3 or 4 lipase elevation occurred in 4.6% of patients in the 90 mg group and 5.5% of patients in the 90→180 mg group. Monitor lipase and amylase during treatment with ALUNBRIG. Withhold ALUNBRIG for Grade 3 or 4 pancreatic enzyme elevation. Upon resolution or recovery to Grade 1 or baseline, resume ALUNBRIG at the same dose or at a reduced dose.

Hyperglycemia: In the ALUNBRIG arm of trial ALTA 1L (180 mg once daily), 56% of patients who received ALUNBRIG experienced new or worsening hyperglycemia. Grade 3 hyperglycemia, based on laboratory assessment of serum fasting glucose levels, occurred in 7.5% of patients. In ALTA, 43% of patients who received ALUNBRIG experienced new or worsening hyperglycemia. Grade 3 hyperglycemia, based on laboratory assessment of serum fasting glucose levels, occurred in 3.7% of patients. Two of 20 (10%) patients with diabetes or glucose intolerance at baseline required initiation of insulin while receiving ALUNBRIG. Assess fasting serum glucose prior to initiation of ALUNBRIG and monitor periodically thereafter. Initiate or optimize anti-hyperglycemic medications as needed. If adequate hyperglycemic control cannot be achieved with optimal medical management, withhold ALUNBRIG until adequate hyperglycemic control is achieved and consider reducing the dose of ALUNBRIG or permanently discontinuing ALUNBRIG.

Embryo-Fetal Toxicity: Based on its mechanism of action and findings in animals, ALUNBRIG can cause fetal harm when administered to pregnant women. There are no clinical data on the use of ALUNBRIG in pregnant women. Advise pregnant women of the potential risk to a fetus. Advise females of reproductive potential to use effective non-hormonal contraception during treatment with ALUNBRIG and for at least 4 months following the final dose. Advise males with female partners of reproductive potential to use effective contraception during treatment and for at least 3 months after the last dose of ALUNBRIG.

ADVERSE REACTIONS

In ALTA 1L, serious adverse reactions occurred in 33% of patients receiving ALUNBRIG. The most common serious adverse reactions other than disease progression were pneumonia (4.4%), ILD/pneumonitis (3.7%), pyrexia (2.9%), dyspnea (2.2%), pulmonary embolism (2.2%), and asthenia (2.2%). Fatal adverse reactions other than disease progression occurred in 2.9% of patients and included pneumonia (1.5%), cerebrovascular accident (0.7%), and multiple organ dysfunction syndrome (0.7%).

In ALTA, serious adverse reactions occurred in 38% of patients in the 90 mg group and 40% of patients in the 90→180 mg group. The most common serious adverse reactions were pneumonia (5.5% overall, 3.7% in the 90 mg group, and 7.3% in the 90→180 mg group) and ILD/pneumonitis (4.6% overall, 1.8% in the 90 mg group and 7.3% in the 90→180 mg group). Fatal adverse reactions occurred in 3.7% of patients and consisted of pneumonia (2 patients), sudden death, dyspnea, respiratory failure, pulmonary embolism, bacterial meningitis and urosepsis (1 patient each).

The most common adverse reactions (≥25%) with ALUNBRIG were diarrhea (49%), fatigue (39%), nausea (39%), rash (38%), cough (37%), myalgia (34%), headache (31%), hypertension (31%), vomiting (27%), and dyspnea (26%).

DRUG INTERACTIONS

CYP3A Inhibitors: Avoid coadministration of ALUNBRIG with strong or moderate CYP3A inhibitors. Avoid grapefruit or grapefruit juice as it may also increase plasma concentrations of brigatinib. If coadministration of a strong or moderate CYP3A inhibitor cannot be avoided, reduce the dose of ALUNBRIG.

CYP3A Inducers: Avoid coadministration of ALUNBRIG with strong or moderate CYP3A inducers. If coadministration of moderate CYP3A inducers cannot be avoided, increase the dose of ALUNBRIG.

CYP3A Substrates: Coadministration of ALUNBRIG with sensitive CYP3A substrates, including hormonal contraceptives, can result in decreased concentrations and loss of efficacy of sensitive CYP3A substrates.

USE IN SPECIFIC POPULATIONS

Pregnancy: ALUNBRIG can cause fetal harm. Advise females of reproductive potential of the potential risk to a fetus.

Lactation: There are no data regarding the secretion of brigatinib in human milk or its effects on the breastfed infant or milk production. Because of the potential adverse reactions in breastfed infants, advise lactating women not to breastfeed during treatment with ALUNBRIG.

Females and Males of Reproductive Potential:

Pregnancy Testing: Verify pregnancy status in females of reproductive potential prior to initiating ALUNBRIG.

Contraception: Advise females of reproductive potential to use effective non-hormonal contraception during treatment with ALUNBRIG and for at least 4 months after the final dose. Advise males with female partners of reproductive potential to use effective contraception during treatment with ALUNBRIG and for at least 3 months after the final dose.

Infertility: ALUNBRIG may cause reduced fertility in males.

Pediatric Use: The safety and effectiveness of ALUNBRIG in pediatric patients have not been established.

Geriatric Use: Of the 359 patients enrolled in the ALTA 1L ALUNBRIG arm and in ALTA, 26.7% were 65 and older and 7.5% were 75 and older. No clinically relevant differences in safety or efficacy were observed between patients ≥65 years and younger patients.

Hepatic or Renal Impairment: No dose adjustment is recommended for patients with mild or moderate hepatic impairment or mild or moderate renal impairment. Reduce the dose of ALUNBRIG for patients with severe hepatic impairment or severe renal impairment.

Please see the full U.S. Prescribing Information for ALUNBRIG at www.ALUNBRIG.com.

Takeda’s Commitment to Oncology

Our core R&D mission is to deliver novel medicines to patients with cancer worldwide through our commitment to science, breakthrough innovation and passion for improving the lives of patients. Whether it’s with our hematology therapies, our robust pipeline, or solid tumor medicines, we aim to stay both innovative and competitive to bring patients the treatments they need. For more information, visit www.takedaoncology.com.

Sensei Biotherapeutics Reports Early Data from Phase 1/2 Clinical Trial of SNS-301 in Combination with Pembrolizumab in Advanced Head and Neck Cancer Patients at ESMO 2020

On September 18, 2020 Sensei Biotherapeutics, Inc., a clinical-stage biopharmaceutical company developing personalized yet off the shelf immunotherapies for cancer and infectious diseases, reported results from the Phase 1/2 clinical trial evaluating the safety, efficacy, and immunogenicity of SNS‑301, a first-in-class bio-engineered, inactivated bacteriophage, in patients with Locally Advanced Unresectable or Metastatic/Recurrent Squamous Cell Carcinoma of the Head and Neck (SCCHN) (Press release, Sensei Biotherapeutics, SEP 18, 2020, View Source [SID1234565362]). The data were presented in a poster discussion session at the European Society for Medical Oncology (ESMO) (Free ESMO Whitepaper) Virtual Congress 2020.

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Patients with SCCHN often present with immune desert or excluded tumors and only 13-16% of the patients respond to anti PD-1/PDL-1 therapy. Data from the KEYNOTE-012 clinical trial demonstrated that SCCHN patients receiving pembrolizumab alone as 2nd line treatment or later have an objective response rate of 18%, median overall survival of 8 months, and progression-free survival of 2 months. Only 6% of patients with PD-L1 negative disease achieve an objective response to pembrolizumab.

"The efficacy of anti-PD-1/PD-L1 is attributed to the presence of infiltrating antigen-specific CD8+ T-cells. Combining anti-PD-1/PD-L1 with agents that generate or expand anti-tumor T-cells, such as vaccines, is critical to increase overall survival of SCCHN patients," said Marie-Louise Fjaellskog, M.D., Ph.D., Chief Medical Officer of Sensei Biotherapeutics. "To date, we have observed promising clinical activity that is correlated with immune response for SNS-301, including a partial response in a patient with PD-L1-negative disease. This initial data from 9 patients provides us with the rationale to continue exploring its safety and efficacy in 1st and 2nd line SCCHN patients."

"We are excited by the emerging translational data for SNS-301, including – in a patient with a confirmed response – a clear conversion from a poorly inflamed tumor into an inflamed microenvironment, characterized by significant T cell infiltration, and upregulation of PD-L1," said Robert Pierce, M.D., Chief Scientific Officer of Sensei Biotherapeutics. "Based on these early promising results, we plan to expand our Immunophage platform to include additional tumor associated antigens and to combine these into bespoke vaccine cocktails based on a patient tumor’s genetic profile."

The ongoing multi-center Phase 1/2 clinical trial of SNS-301 in combination pembrolizumab is designed to assess the safety, efficacy and immunogenicity of SNS-301 in SCCHN patients that had received anti-PD1/PD-L1 therapy for at least 3 months prior to enrollment with stable disease or unconfirmed progressive disease as their best response upon entry into the study.

Highlights of the Safety, Efficacy and Immunogenicity Data as of July 23, 2020 include:

SNS‑301 in combination with pembrolizumab was well tolerated with a favorable safety profile. No dose-limiting toxicities were observed during the safety run-in, and reported adverse events were mostly of Grade 1-2 and considered unrelated to study treatment.
Of the 9 patients evaluable as of the publication date, 6 remain on study. Of these:
One patient with PD-L1 negative disease and stable disease on pembrolizumab upon study entry was converted to a partial response with a tumor reduction of 43%.
One patient with progressive disease upon study entry was converted to stable disease.
Four patients with stable disease upon study entry remain stable, with 2 of these patients on study for 8 months or more.
Immunohistochemical staining of paired pre- and on-treatment biopsies (12 weeks) from the responding patient’s tumor demonstrate a conversion from a PD-L1-negative, poorly inflamed phenotype into an inflamed, PD-L1-positive tumor, characterized by tumor necrosis, and abundant infiltrating immune cells, including CD4, CD8 T cells and macrophages. This patient also demonstrated a significant serological response to SNS-301.
All 29 patients screened for the study were highly positive for the ASPH.
Based on these data, Sensei plans to enroll all 30 patients for this study. An additional study in neoadjuvant SCCHN patients is planned to begin early next year in combination with Imfimzi (durvalumab).

About SNS-301

SNS-301 is a first-in-class cancer immunotherapy designed to overcome immune tolerance and induce robust and durable antigen-specific humoral and cellular responses. It is a bio-engineered, inactivated bacteriophage virus expressing a fusion protein of native bacteriophage GPD (Glyceraldehyde-3-phosphate dehydrogenase) protein and a selected domain of aspartate β-hydroxylase (ASPH). Expression of ASPH is uniquely upregulated in more than 20 different types of cancer and expression levels in various tumors are generally inversely correlated with disease prognosis ASPH signaling is related to cancer cell growth, cell motility and invasiveness, occurs through the Notch pathway, and is implicated in the epithelial to mesenchymal transition (EMT).