European Commission Grants Marketing Authorisation for DARZALEX®▼(daratumumab) Subcutaneous Formulation for all Currently Approved Daratumumab Intravenous Formulation Indications

On June 4, 2020 The Janssen Pharmaceutical Companies of Johnson & Johnson reported that the European Commission (EC) has granted marketing authorisation for DARZALEX▼ (daratumumab) subcutaneous (SC) formulation for the treatment of adult patients with multiple myeloma (MM) (Press release, Johnson & Johnson, JUN 4, 2020, View Source [SID1234560847]). Daratumumab SC is administered as a fixed dose, which significantly reduces treatment time, from hours to approximately three to five minutes, when compared to daratumumab intravenous (IV) formulation.1 In addition, only the first dose of daratumumab SC needs to be administered in an environment where resuscitation facilities are available. The approval applies to all current daratumumab indications in frontline and relapsed/refractory settings, and patients currently on daratumumab IV can switch to the SC formulation should they choose to.

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Data supporting the approval show that daratumumab SC demonstrated a consistent overall response rate (ORR) and a similar safety profile compared with daratumumab IV in patients with relapsed or refractory MM.1 In addition, there was a nearly two-thirds reduction in systemic infusion-related reactions (IRRs) for daratumumab SC compared to daratumumab IV (13 percent vs. 35 percent, respectively).1 The novel SC formulation of daratumumab is co-formulated with recombinant human hyaluronidase PH20 (rHuPH20) [Halozyme’s ENHANZE drug delivery technology].

"Multiple myeloma is an incurable blood cancer that is often associated with time-intensive treatment regimens, which can be burdensome for patients and physicians. Today’s approval marks important progress for the oncology community as it means daratumumab can now be administered in significantly less time, thereby reducing the time patients need to be in the clinical setting," said Maria-Victoria Mateos, M.D., Ph.D., COLUMBA primary investigator and Director of the Myeloma Unit at University Hospital of Salamanca-IBSAL, Salamanca, Spain. "Given the current health climate, this is timely and welcome news, particularly for immunocompromised patients."

"This new formulation was specifically designed as the next step in enhancing the treatment experience with daratumumab, without compromising on safety or efficacy," said Patrick Laroche, M.D., Haematology Therapy Area Lead, Europe, Middle East and Africa (EMEA), Janssen-Cilag. "Since its first launch, daratumumab has been used by more than 130,000 patients globally, and Janssen is pleased to expand our offering by making the subcutaneous formulation available for all previously approved indications."

The approval is supported by data from the Phase 2 PLEIADES (MMY2040) and Phase 3 COLUMBA (MMY3012) studies.

In the PLEIADES study, which evaluated the efficacy and safety of daratumumab SC in combination therapies, objective responses were demonstrated in combination with bortezomib, melphalan, and prednisone (D-VMP) in newly diagnosed transplant ineligible patients.2 In addition, objective responses were demonstrated in combination with lenalidomide and dexamethasone (D-Rd) in relapsed or refractory patients who received one prior line of therapy.2

In the COLUMBA study, at a median follow-up of 7.5 months, the ORR was 41 percent for patients taking daratumumab SC as a monotherapy, compared to 37 percent for those taking daratumumab IV as a monotherapy (95 percent confidence interval [CI], 1.11 (0.89-1.37); P<0.0001).3 The ORR was similar across all clinically relevant subgroups, including bodyweight.1 The ratio of geometric means of Ctrough for daratumumab SC over daratumumab IV was 108 percent (90 percent CI, 96 percent-122 percent).1 The progression-free survival was comparable between the daratumumab SC and daratumumab IV (Hazard Ratio [HR] = 0.99; 95 percent CI, 0.78-1.26; P<0.9258).1 The median duration for each SC injection was five minutes, compared to more than three hours with IV infusions.1

The most common (>5 percent) Grade 3/4 treatment-emergent adverse events (TEAEs) were thrombocytopenia (14 percent vs. 14 percent), anaemia (13 percent vs. 14 percent) and neutropenia (13 percent vs. 8 percent).3 A lower rate of IRRs was observed in the arm that received daratumumab SC compared to daratumumab IV (13 percent vs. 35 percent, respectively) (Odds Ratio = 0.28; 95 percent CI (0.18-0.44); P<0.0001).3 The primary reasons for treatment discontinuation included progressive disease (43 percent in the SC arm vs. 44 percent in the IV arm) and adverse events (7 percent in the SC arm vs. 8 percent in the IV arm).1

"Today’s approval highlights Janssen’s commitment to gaining a better understanding of the evolving needs of people living with multiple myeloma, and to the development of new innovations, combinations, and formulations to best meet those needs," adds Craig Tendler, M.D., Vice President, Clinical Development and Global Medical Affairs, Oncology at Janssen Research & Development, LLC.

Outside of Europe, Janssen recently received approval from the U.S. Food and Drug Administration for the SC formulation of DARZALEX – known locally as DARZALEX FASPRO (daratumumab and hyaluronidase-fihj) – for the treatment of patients with MM.4

#ENDS#

In Europe, daratumumab is indicated:5

in combination with lenalidomide and dexamethasone or with bortezomib, melphalan and prednisone for the treatment of adult patients with newly diagnosed multiple myeloma who are ineligible for autologous stem cell transplant
in combination with bortezomib, thalidomide and dexamethasone for the treatment of adult patients with newly diagnosed multiple myeloma who are eligible for autologous stem cell transplant
in combination with lenalidomide and dexamethasone, or bortezomib and dexamethasone, for the treatment of adult patients with multiple myeloma who have received at least one prior therapy
as monotherapy for the treatment of adult patients with relapsed and refractory multiple myeloma, whose prior therapy included a proteasome inhibitor and an immunomodulatory agent and who have demonstrated disease progression on the last therapy
About the COLUMBA Study (MMY3012)6,7

The randomised, open-label, multicentre Phase 3 study included 522 patients with multiple myeloma (MM) who had received at least three prior lines of therapy including a proteasome inhibitor (PI) and an immunomodulatory drug (IMiD), or whose disease was refractory to both a PI and an IMiD. In the arm that received daratumumab subcutaneous formulation (SC; n=263), patients (median age of 65) received a fixed dose of daratumumab 1,800 milligrams (mg) co-formulated with recombinant human hyaluronidase (rHuPH20) 2,000 Units per millilitre (U/mL), SC weekly for cycles 1 – 2, every two weeks for cycles 3 – 6, and every four weeks for cycle 7 and thereafter. In the arm that received daratumumab intravenous formulation (IV; n=259), patients (median age of 67) received 16 milligrams per kilogram (mg/kg) weekly for cycles 1 – 2, every two weeks for cycles 3 – 6, and every four weeks for cycle 7 and thereafter. Each cycle was 28 days. Patients in both treatment arms continued until disease progression or unacceptable toxicity. Co-primary endpoints were overall response rate (ORR; non-inferiority = 60 percent retention of the lower bound [20.8 percent] of the 95 percent confidence interval [CI] of the SIRIUS trial, with relative risk [RR] analysed by Farrington-Manning test) and pre-dose cycle 3, day 1 (C3D1) daratumumab Ctrough (non-inferiority = lower bound of 90 percent CI for the ratio of the geometric means [GM] ≥80 percent).

About the PLEIADES Study (MMY2040)8

The non-randomised, open-label, parallel assignment Phase 2 PLEIADES trial included 240 adults either newly diagnosed or with relapsed or refractory multiple myeloma (MM). Patients with newly diagnosed MM were treated with 1,800 mg of daratumumab subcutaneous formulation (SC) in combination with either bortezomib, lenalidomide and dexamethasone (D-VRd) or bortezomib, melphalan and prednisone (D-VMP). Patients with relapsed or refractory disease were treated with 1,800 mg of daratumumab SC plus lenalidomide and dexamethasone (D-Rd). The primary endpoint for the D-VMP and D-Rd cohorts was overall response rate (ORR). The primary endpoint for the D-VRd cohort was very good partial response or better rate. An additional cohort of patients with relapsed and refractory MM treated with daratumumab SC plus carfilzomib and dexamethasone was subsequently added to the study.

About daratumumab

Daratumumab is a first-in-class9 biologic targeting CD38, a surface protein that is highly expressed across multiple myeloma (MM) cells, regardless of disease stage.10 Daratumumab is believed to induce tumour cell death through multiple immune-mediated mechanisms of action, including complement-dependent cytotoxicity (CDC), antibody-dependent cell-mediated cytotoxicity (ADCC) and antibody-dependent cellular phagocytosis (ADCP), as well as through apoptosis, in which a series of molecular steps in a cell lead to its death.5 A subset of myeloid derived suppressor cells (CD38+ MDSCs), CD38+ regulatory T cells (Tregs) and CD38+ B cells (Bregs) are decreased by daratumumab-mediated cell lysis.5 Since launch, it is estimated that 130,000 patients have been treated with daratumumab worldwide.11 Daratumumab is being evaluated in a comprehensive clinical development programme across a range of treatment settings in MM, such as in frontline and relapsed settings.12,13,14,15,16,17,18,19 Additional studies are ongoing or planned to assess its potential in other malignant and pre-malignant haematologic diseases in which CD38 is expressed, such as smouldering myeloma.20,21 For more information, please see View Source

For further information on daratumumab, please see the Summary of Product Characteristics at View Source

In August 2012, Janssen Biotech, Inc. and Genmab A/S entered a worldwide agreement, which granted Janssen an exclusive licence to develop, manufacture and commercialise daratumumab.22

About Multiple Myeloma (MM)

Multiple myeloma (MM) is an incurable blood cancer that starts in the bone marrow and is characterised by an excessive proliferation of plasma cells.23 In Europe, more than 48,200 people were diagnosed with MM in 2018, and more than 30,800 patients died.24 Around 50 percent of newly diagnosed patients do not reach five-year survival,25,26 and almost 29 percent of patients with multiple myeloma will die within one year of diagnosis.27

Although treatment may result in remission, unfortunately, patients will most likely relapse as there is currently no cure.28 Relapsed and refractory myeloma is defined as disease that is nonresponsive while on salvage therapy, or progresses within 60 days of last therapy in patients who have achieved minimal response (MR) or better at some point previously before then progressing in their disease course.29 While some patients with MM have no symptoms at all, others are diagnosed due to symptoms that can include bone problems, low blood counts, calcium elevation, kidney problems or infections.30 Patients who relapse after treatment with standard therapies, including proteasome inhibitors and immunomodulatory agents, have poor prognoses and require new therapies for continued disease control.31

TumorCube, a New Unit within DiscoveryBioMed, Will Use 3D Biogel-based Platforms to Perform 384-Well Plate-based Bioassays and 3D Bioreactor-driven Studies to Discover, Validate and Profile Anti-cancer Therapeutics on Human Cancer Cells

On June 4, 2020 DiscoveryBioMed, Inc. (DBM, www.discoverybiomed.com) reported a new unit of its contract research organization (CRO) services business called TumorCube that is formed based upon our growing expertise in 3D Biogel-driven bioassays on diseased and normal human cell platform technologies (Press release, DiscoveryBioMed, JUN 4, 2020, View Source [SID1234560846]). A trademark application has been filed.

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"DBM has a growing CRO services business in 3D Biogel-driven bioassays of diseased and normal human kidney cells. It is showcased prominently on our website, and we are proud to have many biotechnology companies and BioPharmaceutical organizations globally as our clients," explained Dr. Erik Schwiebert, Ph.D., CEO of DBM. "We have now extended those offerings to include the assay of human urological cancers and endocrine-driven cancers such as ovarian cancers and breast cancers." DBM is also assessing the feasibility of our 3D platforms for the assay of neurological and other cancers at present. DBM is utilizing existing human cancer cell lines in our initial technology optimization.

"Interestingly, diseased human kidney cells and existing human cancer cell lines respond differently to specific lead therapeutic drugs or industry standard anti-cancer drugs when grown in 3D versus the traditional 2D tissue culture plastic-based assays," explained Dr. Deborah Mai, DBM’s Chief Biological Officer. "DBM focuses on more physiological 3D growth formats as its preferred assay platforms and methods."

Not only does DBM wish to offer higher-throughput 3D assay platforms for hit-to-lead and lead oncology therapeutic profiling, DBM is optioning and licensing 3D bioreactor technology from the University of Alabama at Birmingham (UAB) that was invented by Dr. Joel Berry, Ph.D. in the Department of Biomedical Engineering to be a future platform for our TumorCube unit. "It has been exciting to collaborate with DBM on this commercial-academic partnership, and we are also excited to see TumorCube realized," declared Dr. Berry. "We and DBM are also collaborating with investigators within the O’Neal Comprehensive Cancer Center at UAB on this platform development, which is an additional value add."

DBM is also pleased about continuing its local collaboration with UAB. "DBM has several current and past UAB-derived scientists that have contributed to the establishment and growth of our life sciences and biotechnology company, myself included," explained Dr. Grace Salzer, a Senior Scientist and Business Development specialist. "DBM is fortunate to be collaborating with UAB scientists and technology transfer on this venture."

AmorChem Invests in the Work of Sidong Huang at McGill University Using a Novel Target to Address a SMARCA4-deficient Rare and Aggressive Form of Ovarian Cancer

On June 4, 2020 AmorChem II L.P. ("AmorChem") reported the closing of a new transaction with McGill University ("McGill"), focusing on the work of Dr. Sidong Huang and his collaborator, Dr. Martin Schmeing, from the Rosalind and Morris Goodman Cancer Research Centre (Press release, Amorchem, JUN 4, 2020, View Source [SID1234560845]). The project, financed by AmorChem, will focus on the identification of small molecules to treat small cell carcinoma of the ovary, hypercalcemic type ("SCCOHT").

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"We were impressed by the sophisticated screening campaigns utilized by Dr. Huang to uncover synthetic lethal targets in various cancers. His focus on the SWI/SNF chromatin remodeling complex was particularly interesting, as it frequently incurs inactivating mutations and deletions in various components of the complex, which are themselves not druggable. His screen identified targets, such as SL2, that expose the vulnerabilities of such mutations and deletions indirectly. SL2 represents an exciting and novel mitochondrial target to treat certain SMARCA4-deficient cancers," says Kevin McBride, Chief Scientific Officer and Partner at AmorChem.

Through this investment, the forces of NuChem Therapeutics inc. ("NuChem") will join with those of the McGill team to identify and optimise small molecule inhibitors of SL2. AmorChem collaborations with NuChem have been successful in the past and the combination of strong medicinal chemistry and assay development teams with a strong functional genomics group should be very positive for the outcome of this project.

"SCCOHT is an aggressive, rare form of ovarian cancer which affects predominantly women in their 20s. With 65% of patients dying within two years, this severe disease represents a definite unmet medical need. If the usefulness of targeting SL2 with small molecules can be proven in SCCOHT, we believe it will also be possible to extend the use of its inhibitors to other oncology indications, such as non-small cell lung cancer," says Elizabeth Douville, Managing Partner at AmorChem.

"I would like to thank AmorChem and NuChem for their investment in Dr. Huang’s work," says Sylvain Coulombe, Associate Vice-Principal, Innovation and Partnerships (I+P), McGill University. "Helping move ground-breaking research from the lab to the clinic – particularly in the service of treating rare, devastating diseases – demonstrates the power of diverse partners working toward a common goal. That is why supporting and promoting research with strong potential for commercialization is and will remain at the core of our mission in I+P and at McGill."

Noxopharm Reports Veyonda’s Potential in Late-Stage Cancer at ASCO 2020

On June 4, 2020 At the 2020 American Society of Clinical Oncology (ASCO) (Free ASCO Whitepaper) Virtual Annual Meeting, Noxopharm, a clinical-stage Australian oncology drug development company, has reported two sets of clinical data relating to the development of NOX66 (Veyonda) as a treatment for end-stage cancer (Press release, Noxopharm, JUN 4, 2020, View Source [SID1234560844]).

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The posters addressed how NOX66 may improve treatment responses in men with mCRPC and also its contribution to restoring sensitivity to apoptosis and potentially modulating the immune microenvironment of nasopharyngeal carcinoma (NPC).

"We have been unaware of any therapies under development that come close to offering an anticancer effect to anything like the same degree that we are seeing with Veyonda, and in particular, delivering this level of benefit in a well-tolerated, minimally invasive, and cost-effective manner," said Dr. Graham Kelly, Noxopharm CEO. "In the context of what has been reported at ASCO (Free ASCO Whitepaper) 2020, the high response rates we are seeing with Veyonda mark it as a major drug prospect."

An estimated 300,000 men die worldwide each year from prostate cancer after exhausting available treatment options. Add to that the generally high pain levels associated with the typical spread of prostate cancer to bone, and the need for a last-line treatment offering a meaningful effect once everything else has failed becomes compelling.

"This is an exciting outcome that supports our belief in the anticancer properties of Veyonda," Dr. Kelly said. "To our knowledge, this is the first time that anyone has been able to obtain a meaningful abscopal response rate in prostate cancer. Prostate cancer has developed a reputation as a cancer with poor immune responsiveness, but this data suggests that this isn’t the case. Today’s result positions Veyonda at the forefront of this emerging area of oncology and suggests that we have an exciting new prospective treatment for end-stage prostate cancer."

U.S. FDA Accepts PharmaEssentia’s Application for Ropeginterferon Alfa-2b to Treat Polythycemia Vera

On June 4, 2020 PharmaEssentia Corporation (TPEx: 6446), a global biopharmaceutical innovator leveraging deep expertise and proven scientific principles to deliver new biologics in hematology and oncology, reported that the U.S. Food and Drug Administration (FDA) has recently accepted its Biologics License Application (BLA) for ropeginterferon alfa-2b (P1101), a novel pegylated interferon intended for the treatment of the rare blood cancer polycythemia vera (PV) in the absence of symptomatic splenomegaly (Press release, PharmaEssentia, JUN 4, 2020, View Source [SID1234560843]). The company expects an agency decision in early 2021.

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PharmaEssentia has focused its efforts on therapeutic innovation in the category of myeloproliferative neoplasms (MPNs), which are caused by specific genetic mutations that lead to overproduction of blood components including white or red blood cells, or platelets. In PV, which is caused by a JAK2 V617F mutation, the bone marrow produces excessive red blood cells, causing the blood to be thicker than normal and potentially leading to a range of complications.1,2 PV is estimated to affect more than 160,000 people in the U.S. alone,1 who have progressively burdensome symptoms. Without proper management, the disease progresses into malignancies including myelofibrosis and acute myeloid leukemia.3

Ropeginterferon alfa-2b was invented by scientists at PharmaEssentia in Taiwan and is a structurally novel monopegylated proline interferon designed for administration once every two weeks. The U.S. filing is supported by robust, durable 24-36-month data from the Phase 3 PROUD/CONTI-PV clinical trial, which demonstrated that the investigational treatment offered high and durable hematologic responses and symptom control with good tolerability and low rates of depression observed, with effects on relevant MPN mutations supporting a potential disease modifying capability.4

The findings were shown among patients who received either Ropeginterferon alfa-2b (n=95) or hydroxyurea/best available therapy (HU/BAT, n=74). At 36 months of treatment, patients who received Ropeginterferon alfa-2b maintained a complete hematological response longer than those who received HU/BAT (70.5% vs. 51.4%). Response rates steadily increased in the Ropeginterferon alfa-2b arm throughout 24 months of treatment and remained constant after 36 months. Further, after 36 months, two thirds (66.0%) of patients who received Ropeginterferon alfa-2b achieved a molecular response, compared with 27% in the HU/BAT arm. Importantly, these molecular responses were closely related to complete hematological responses. There were similar rates of adverse events in both arms; the most common (>10%) treatment-related adverse events included anemia, thrombocytopenia and leukopenia, which occurred more frequently under HU.4

"Our focus is on stunting these rare malignancies, preserving patient well-being and slowing the progression into more aggressive and deadly cancers," said Meredith Manning, U.S. General Manager for PharmaEssentia. "We believe Ropeginterferon alfa-2b could become an important new therapeutic tool and look forward to engaging with the regulators in our efforts to introduce this option to the underserved PV community in the U.S."

Ms. Manning was recently appointed to the U.S. GM role to guide the expansion of the company’s U.S. presence, with near-term focus on the commercial preparations for the first target indication in PV. Ms. Manning brings PharmaEssentia dynamic expertise in commercialization and market strategy. She joined from resTORbio, where she served as Chief Commercial Officer to define the corporate strategy and the commercial launch approach for an aging-related therapeutic.

About Ropeginterferon alfa-2b

Ropeginterferon alfa-2b is a novel, long-acting, mono-pegylated proline interferon that has been engineered with an optimized profile to support improved pharmacokinetic properties and demonstrated tolerability and convenience compared with conventional interferons. It is designed for administration once every two weeks, or once every four weeks during long-term maintenance. Ropeginterferon alfa-2b has Orphan Drug designation for treatment of polycythemia vera (PV) in the United States. Marketed as Besremi in Europe, the product was approved by the European Medicines Agency (EMA) in 2019. Ropeginterferon alfa-2b was discovered and is manufactured by PharmaEssentia in its Taichung plant, which was cGMP certified by TFDA in 2017 and by EMA in January 2018.

About Polycythemia Vera

Polycythemia Vera (PV) is a cancer originating from a disease-initiating stem cell in the bone marrow resulting in a chronic increase of red blood cells, white blood cells, and platelets. This condition may result in cardiovascular complications such as thrombosis and embolism, as well as transformation to secondary myelofibrosis or leukemia. While the molecular mechanism underlying PV is still subject of intense research, current results point to a set of acquired mutations, the most important being a mutant form of JAK2.3