Provectus Announces Preliminary Results from Phase 1 Trial of PV-10 for Treatment of Metastatic Neuroendocrine Tumors at ASCO 2019

On June 3, 2019 Provectus (OTCQB: PVCT) reported that preliminary safety, response, biomarker, and quality of life results from the Company’s ongoing Phase 1 clinical trial of single agent PV-10 for the treatment of symptomatic neuroendocrine tumors (NET) metastatic to the liver (mNET) were presented at the American Society of Clinical Oncology (ASCO) (Free ASCO Whitepaper) 2019 Annual Meeting, held in Chicago, IL from May 31-June 4, 2019 (Press release, Provectus Biopharmaceuticals, JUN 3, 2019, View Source [SID1234536830]). Intralesional injection of oncolytic immunotherapy PV-10 can yield immunogenic cell death in solid tumor cancers that results in tumor-specific reactivity in circulating T cells.1-5 PV-10 clinical development includes cutaneous melanoma, hepatocellular carcinoma, and metastatic liver cancers such as uveal melanoma in single-agent and combination therapy settings.

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This single-center Phase 1 study is being conducted at The Queen Elizabeth Hospital in Adelaide, Australia to evaluate the potential safety, tolerability, and preliminary efficacy of PV-10 in mNET patients (NCT02693067). The primary endpoint for the trial is safety, and secondary endpoints include objective response rate (ORR) of injected target and measurable bystander lesions, target lesion somatostatin receptor expression, and biochemical response. Six patients in the first cohort each received one percutaneously-administered injection of PV-10 to one target lesion per treatment cycle. Patients in the second cohort can receive PV-10 injection of multiple lesions per cycle (2 of 6 patients in the second cohort have received at least one cycle of PV-10 thus far).

Preliminary Results from the Presentation at ASCO (Free ASCO Whitepaper):

Baseline characteristics (N=6): 67% men; median age of 65 years (range 47-72).

Disease characteristics: primary tumor site – 50% small intestine, 33% pancreas, and 17% caecal; 87% Grade 2 (well differentiated, intermediate); all patients were refractory to systemic somatostatin analogues and peptide receptor radionuclide therapy.

PV-10 treatment summary: Median of 1 cycle (mean 1.7, range 1-4) and median dose per cycle of 2.1 mL (range 1.0-5.8 mL).

Preliminary safety: Acceptable toxicity (e.g., post-procedure pain, carcinoid flare, nausea); liver function tests have remained stable.

Preliminary target lesion efficacy: 50% objective response and 87% disease control; response follow-up in 3 patients (50%) is ongoing

Preliminary patient-level efficacy data were not available at the time of data cutoff.

Preliminary clinical and biomarker outcomes: overall quality of life scores were stable in 5 patients (87%); Chromogranin A responses were stable in 5 patients (87%)

Preliminary changes in peripheral blood mononuclear cells were not available at the time of data cutoff.
Dominic Rodrigues, Vice Chair of the Company’s Board of Directors, said, "Patients with neuroendocrine tumors remain a cancer population with high unmet medical need. This poster describes Provectus’ continued progress towards demonstrating the widespread use of small molecule oncolytic immunotherapy PV-10 for the treatment of immunologically ‘cold’ or non-T cell inflamed tumor types."

Mr. Rodrigues added, "Patients in this study experienced no safety concerns from single or repeated injections of PV-10 into the liver. Furthermore, we believe PV-10 treatment provided in this trial yielded encouraging, preliminary evidence of single-agent drug activity, including both local and systemic disease control."

A copy of the ASCO (Free ASCO Whitepaper) poster presentation is currently available on Provectus’ website at View Source

About Neuroendocrine Tumors

NET associated with the gastrointestinal tract are frequently indolent but troublesome as a result of endocrine secretory properties and a propensity for metastasis to the liver, nodes, and lungs. mNET located in the midgut and liver often secrete vasoactive products, giving rise to "Carcinoid Syndrome" (e.g., flushing, diarrhea, wheezing, abdominal cramps, and peripheral edema).

About PV-10

PV-10 causes acute oncolytic destruction of injected tumors, releasing damage associated molecular pattern molecules (DAMPs) and tumor antigens that initiate an immunologic cascade where local response by the innate immune system facilitates systemic anti-tumor immunity by the adaptive immune system. The DAMP release-mediated adaptive immune response activates lymphocytes, including CD8+ T cells, CD4+ T cells, and NKT cells, based on clinical and preclinical experience in multiple tumor types. T cell function can be further augmented by combining PV-10 with immune checkpoint inhibition.

PV-10 is undergoing clinical study for adult solid tumor cancers like melanoma and cancers of the liver (including metastatic symptomatic neuroendocrine tumors and metastatic uveal melanoma) and preclinical study for pediatric cancers like neuroblastoma5, Ewing sarcoma, rhabdomyosarcoma, and osteosarcoma.

Orphan drug designation status has been granted to PV-10 by the U.S. Food and Drug Administration for the treatments of metastatic melanoma in 2006, hepatocellular carcinoma in 2011, neuroblastoma in 2018, and ocular melanoma (including uveal melanoma) in 2019.

PV-10’s active pharmaceutical ingredient is rose bengal disodium (RB) (4,5,6,7-tetrachloro-2’,4’,5’,7’-tetraiodofluorescein disodium salt), a small molecule halogenated xanthene. PV-10 drug product is a bright rose red solution containing 10% w/v RB in 0.9% saline for injection, which is supplied in single-use glass vials containing 5 mL (to deliver) of solution and administered without dilution to solid tumors via intratumoral injection.

Provectus’ intellectual property includes a family of US and international patents that protect the process by which pharmaceutical grade RB and related xanthenes are produced, reducing the formation of previously unknown transhalogenated impurities that exist in commercial grade RB in uncontrolled amounts. The requirement to identify and control these substances is in accordance with International Conference on Harmonisation (ICH) guidelines for the manufacturing of active pharmaceutical ingredient that is suitable for clinical trial and commercial pharmaceutical use. US patent numbers are 8,530,675, 9,273,022, and 9,422,260; patent expirations range from 2030 to 2031.

NantHealth and NantOmics to Present Data on Tumor Mutation Burden (TMB) and PD-L1 Expression in SDH/FH Mutated Solid Tumors at the American Society of Clinical Oncology (ASCO) 2019 Annual Meeting

On June 3, 2019 NantWorks, LLC reported that its affiliate companies, NantHealth, Inc., (NASDAQ: NH), a leading next-generation, evidence-based, personalized healthcare company, and NantOmics, LLC, the leader in molecular analyses , will present data on the association between increased TMB and increased PD-L1 expression with the presence of SDH/FH mutations in a variety of tumors, using 3,461 paired tumor/normal whole exome sequences from the NantHealth clinical cases database to look into the potential therapeutic role for inhibition of PD-1/PD-L1 pathway in these tumors during the cancer prevention, hereditary genetics and epidemiology session at the ASCO (Free ASCO Whitepaper) 2019 Annual Meeting, an event bringing together more than 32,000 global oncology professionals from May 31-June 4, 2019 at McCormick Place in Chicago, Illinois (Press release, NantHealth, JUN 3, 2019, View Source [SID1234536828]). NantHealth and NantOmics conducted this study with researchers from Virginia Commonwealth University’s Massey Cancer Center. NantWorks will be exhibiting at booth #24080 during the event.

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"We believe the data from our retrospective analysis may impact the use of immunotherapy for SDH/FH deficient tumors," said Sandeep "Bobby" Reddy, MD, Chief Medical Officer, NantHealth. "Our analysis provides actionable insight and evidence-based support to further our mission of optimizing patient outcomes and enabling value-based care, specifically in the oncology realm."

Presentation Details

Tumor mutation burden and PD-L1 expression in SDH/FH mutated solid tumors, Abstract #1524
WHO:NantHealth, Inc. and NantOmics, LLC
WHAT: Cancer Prevention, Hereditary Genetics and Epidemiology Session
WHEN:June 3, 1:15-4:15 PM CDT
WHERE: Hall A, McCormick Place

Presentation Summary

Succinate Dehydrogenases and Fumarate Hydratase (SDH/FH) deficient tumors are characterized by succinate/fumarate accumulation and resultant pseudohypoxia that drives malignant transformation. It has been recently shown that HIF-1a stabilization due to hypoxia can lead to upregulation of PD-1 ligand. This study explored tumor mutation burden (TMB), gene expression of PD-L1 and expression of other immune checkpoint-associated genes in a diverse cohort of human tumors harboring SDH A, B, C, D and FH mutations. Retrospective analysis was performed on 3,461 paired tumor/normal whole exome sequences (WES; ~150x coverage) from the NantHealth clinical cases database. 42 clinical cases with potentially pathogenic variants (pPV) in SDHx and FH were identified. Variant pathogenicity was assessed by multiple factors including driver gene status, variant class (e.g. Missense), PhastCons conservation score and population allele frequency. The study found an association between increased TMB and increased PD-L1 expression with the presence of SDH/FH mutations in a variety of tumors. These key parameters imply that a higher TMB may drive the evolutionary pressure to select clones with a PD-L1 high phenotype. This observation supports a potential therapeutic role for inhibition of PD-1/PD-L1 pathway in these tumors.

Blueprint Medicines’ Highly Selective RET Inhibitor BLU-667 Shows Durable Anti-Tumor Activity in Patients with RET-Altered Cancers in Updated ARROW Trial Data Presented at ASCO 2019

On June 3, 2019 Blueprint Medicines Corporation (NASDAQ: BPMC), a precision therapy company focused on genomically defined cancers, rare diseases and cancer immunotherapy, reported updated data from the ongoing registration-enabling ARROW trial of BLU-667 in patients with RET-altered cancers (Press release, Blueprint Medicines, JUN 3, 2019, View Source [SID1234536826]). The data presented at the American Society of Clinical Oncology (ASCO) (Free ASCO Whitepaper) 2019 Annual Meeting show durable clinical activity in patients with RET-altered non-small cell lung cancer (NSCLC), medullary thyroid cancer (MTC) and other cancers. Designed by Blueprint Medicines, BLU-667 is a potent and highly selective oral inhibitor of RET fusions and mutations, including predicted resistance mutations.

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The new results support Blueprint Medicines’ plans to submit an initial New Drug Application (NDA) to the U.S. Food and Drug Administration (FDA) for BLU-667 for the treatment of patients with RET-fusion NSCLC previously treated with platinum-based chemotherapy in the first quarter of 2020, and an NDA to the FDA for the treatment of patients with RET-mutant MTC previously treated with an approved multi-kinase inhibitor (MKI) in the first half of 2020.

"Targeted therapies have transformed the management of multiple oncogenic subsets of lung cancer, but RET-fusion positive lung cancers have not derived similar benefit from current therapeutic approaches. To date, no selective RET inhibitors are approved," said Justin Gainor, M.D., director of Targeted Immunotherapy at Massachusetts General Hospital Cancer Center and an investigator on the ARROW trial. "In the data presented at ASCO (Free ASCO Whitepaper), BLU-667 demonstrated high response rates across multiple populations of RET-altered cancer patients, including patients with untreated brain metastases."

"This growing body of evidence supports our plans to rapidly advance BLU-667, a highly selective RET inhibitor, for the treatment of patients with RET-altered cancers," said Andy Boral, M.D., Ph.D., Chief Medical Officer at Blueprint Medicines. "We remain on track to submit our first New Drug Application to the FDA for BLU-667 for previously treated RET-fusion non-small cell lung cancer patients in the first quarter of 2020. Based on the encouraging data to date and FDA feedback, we are now working to expand ARROW trial enrollment for treatment-naive patients with RET-fusion positive non-small cell lung cancer, with the goal of supporting an accelerated path to registration in first-line patients. In addition, based on the strong data for BLU-667 across RET alteration and tumor types, we plan to continue to work with investigators and global regulatory authorities to bring BLU-667 to the broader population of patients with RET-altered cancers who could potentially benefit from this treatment."

Highlights from ASCO (Free ASCO Whitepaper) Presentations of ARROW Trial Data

The presented data include 120 patients with RET-fusion NSCLC, 64 patients with RET-mutant MTC and 12 patients with other RET-altered cancers (nine papillary thyroid cancer (PTC), two pancreatic cancer and one intrahepatic bile duct carcinoma) enrolled in the ARROW trial as of a data cutoff date of April 28, 2019. The patients with RET-fusion NSCLC and RET-mutant MTC received a starting dose of 400 mg once daily (QD), which is the recommended Phase 2 dose (RP2D). Patients with other RET-altered cancers were included regardless of starting dose.

At baseline, 40 percent of the RET-fusion NSCLC patients had brain metastases. Brain metastases commonly occur in NSCLC patients, and the prognosis in these patients is typically poor. Regardless of starting dose and including the dose-escalation portion of the ARROW trial, the RET-fusion NSCLC patients have been on treatment up to 24 months.

For clinical activity data, NSCLC and MTC patients were evaluable if they were enrolled as of November 14, 2018 with follow-up through the data cutoff date, which enabled them to have at least two radiographic scans. Tumor response was assessed by Response Evaluation Criteria in Solid Tumors (RECIST) version 1.1.

Clinical Activity Data — RET-Fusion NSCLC

As of the data cutoff date, 48 patients with RET-fusion NSCLC were evaluable for response assessment, including 35 patients previously treated with platinum-based chemotherapy.

Nearly all patients (90 percent) had radiographic tumor reductions.
The objective response rate (ORR) was 60 percent (one complete response and 20 partial responses (PR); all responses were confirmed), and the disease control rate1 (DCR) was 100 percent in the patients previously treated with platinum-based chemotherapy.
The ORR was 71 percent (five confirmed PRs) in seven patients naïve to prior systemic treatment.
Across all patients, the median duration of response (DOR) was not reached, and 82 percent of responders remained on treatment as of the data cutoff date.
In nine patients with measurable brain metastases, 78 percent had shrinkage of brain metastases.
No patients starting at the 400 mg QD dose had disease progression due to new brain involvement.
BLU-667 was highly active regardless of RET fusion partner, including RET-KIF5B and RET-CCDC6.
Clinical Activity Data — RET-Mutant MTC and Other RET-Altered Cancers

As of the data cutoff date, 32 patients with RET-mutant MTC were evaluable for response assessment, including 16 patients previously treated with the MKIs cabozantinib or vandetanib.

The ORR was 63 percent (nine confirmed PRs, one PR pending confirmation) and the DCR was 94 percent in RET-mutant MTC patients previously treated with cabozantinib or vandetanib.
Across all RET-mutant MTC patients, the median DOR was not reached and all responders remained on treatment as of the data cutoff date, with treatment durations up to 15.6 months for patients receiving a starting dose of 400 mg QD.
As of the data cutoff date, clinical activity data were reported in patients with other RET-altered cancers:

Six patients with PTC were evaluable for response assessment by RECIST version 1.1. In these patients, the ORR was 83 percent (three confirmed PRs, two PRs pending confirmation).
Five patients with PTC have remained on treatment for one year or longer, and eight patients with PTC remained on treatment as of the data cutoff date.
Additional responses were observed in patients with other RET-fusion cancers, including pancreatic cancer (one confirmed PR, one PR pending confirmation) and intrahepatic bile duct carcinoma (one confirmed PR).
Four patients (two with RET-fusion NSCLC, two with RET-mutant MTC) enrolled in the ARROW trial were previously treated with LOXO-292. Among them:

Two patients had a PR, one of which was confirmed as of the data cutoff date, and one of which was pending as of the data cutoff date and subsequently confirmed prior to the presentation.
One patient had stable disease with radiographic tumor reductions and remained on treatment as of the data cutoff date.
Safety Data

As of the data cutoff date, 226 patients received a starting dose of 400 mg QD and were evaluable for safety. Across all patients, BLU-667 was well-tolerated and most adverse events (AEs) reported by investigators were Grade 1 or 2. Across all grades, the most common treatment-emergent AEs (regardless of relationship to BLU-667) reported by investigators (≥15 percent) were constipation, hypertension, increased aspartate aminotransferase, neutropenia, diarrhea, fatigue, anemia, increased alanine aminotransferase and increased blood creatinine. Investigator-reported Grade 3 or 4 treatment-related AEs (≥2 percent) included neutropenia, hypertension, anemia, increased blood creatine phosphokinase and leukopenia.

Across all patients, only 4 percent of patients discontinued treatment with BLU-667 due to treatment-related AEs. Seven percent of patients with RET-fusion NSCLC discontinued treatment with BLU-667 due to treatment-related AEs, and no patients with RET-mutant MTC discontinued treatment with BLU-667 due to treatment-related AEs.

These updated data for BLU-667 were reported in two presentations at the ASCO (Free ASCO Whitepaper) 2019 Annual Meeting, including a poster presentation on trial results in thyroid cancer on Saturday, June 1 (Abstract Number: 6018) and an oral presentation on trial results in NSCLC on Monday, June 3 (Abstract Number: 9008). Copies of the data presentations are available in the "Science—Publications and Presentations" section of Blueprint Medicines’ website at www.BlueprintMedicines.com.

BLU-667 Clinical Development Update

Based on encouraging clinical activity in patients with NSCLC naïve to prior systemic therapy and feedback from the FDA, Blueprint Medicines reported plans to expand the enrollment target of the ongoing ARROW trial cohort for treatment-naïve patients with RET-fusion NSCLC, with the goal of supporting expedited development in first-line RET-fusion NSCLC.

Investor Event and Webcast Information

Blueprint Medicines will host an investor event on Monday, June 3, 2019 beginning at 6:00 p.m. CT (7:00 p.m. ET) in Chicago to provide a portfolio update, including a review of updated clinical data from the ongoing ARROW trial of BLU-667 in patients with RET-altered cancers and the ongoing registration-enabling NAVIGATOR trial in patients with PDGFRA Exon 18 mutant and fourth-line gastrointestinal stromal tumors (GIST). Formal presentations and the live webcast will begin at 6:30 p.m. CT (7:30 p.m. ET). The event will be webcast live and can be accessed under the "Investors & Media—Events & Presentations" section of Blueprint Medicines’ website at www.BlueprintMedicines.com. A replay of the webcast will be available approximately two hours after the event and will be available for 30 days following the event.

About the ARROW Trial

ARROW is a Phase 1 clinical trial designed to evaluate the safety, tolerability and efficacy of BLU-667 in multiple ascending doses in adults with RET-altered NSCLC, MTC and other advanced solid tumors. The trial consists of two parts: a dose escalation portion, which is now complete, and an expansion portion, in which enrollment is ongoing. The expansion portion consists of seven defined cohorts of patients treated with BLU-667 at the RP2D of 400 mg QD: (1) RET-fusion NSCLC patients previously treated with a platinum-based chemotherapy, (2) RET-fusion NSCLC patients who have not previously received a platinum-based chemotherapy, (3) RET-mutant MTC patients previously treated with cabozantinib or vandetanib, (4) RET-mutant MTC patients who have not previously received cabozantinib or vandetanib, (5) patients with other RET-fusion tumors, (6) patients with other RET-mutant tumors and (7) RET-altered solid tumor patients previously treated with a selective RET inhibitor. Trial objectives include assessing response, pharmacokinetics, pharmacodynamics and safety. The trial is enrolling patients at multiple sites in the United States, European Union and Asia.

Patients and physicians interested in the ARROW clinical trial can contact the Blueprint Medicines study director at [email protected] or 1-617-714-6707. Additional details are available at www.BlueprintClinicalTrials.com/ARROW or www.clinicaltrials.gov (ClinicalTrials.gov Identifier: NCT03037385).

About RET-Altered Solid Tumors

RET activating fusions and mutations are key disease drivers in many cancer types, including NSCLC and MTC. RET fusions are implicated in approximately 1 to 2 percent of patients with NSCLC and approximately 10 to 20 percent of patients with PTC, while RET mutations are implicated in approximately 90 percent of patients with advanced MTC. In addition, oncogenic RET alterations are observed at low frequencies in colorectal, breast, pancreatic and other cancers, and RET fusions have been observed in patients with treatment-resistant, EGFR-mutant NSCLC.

Currently, there are no approved therapies that selectively target RET-driven cancers, although there are several approved MKIs with RET activity being evaluated in clinical trials. To date, clinical activity attributable to RET inhibition has been uncertain for these approved MKIs, likely due to insufficient inhibition of RET and off-target toxicities. There is a need for precision therapies that provide durable clinical benefit by selectively targeting RET alterations and anticipated resistance mutations.

About BLU-667

BLU-667 is an investigational, once-daily oral precision therapy specifically designed for highly potent and selective targeting of oncogenic RET alterations. Blueprint Medicines is developing BLU-667 for the treatment of patients with RET-altered NSCLC, MTC and other solid tumors. The FDA has granted Breakthrough Therapy Designation to BLU-667 for the treatment of RET-fusion positive NSCLC that has progressed following platinum-based chemotherapy, and RET-mutation positive MTC that requires systemic treatment and for which there are no acceptable alternative treatments.

BLU-667 was designed by Blueprint Medicines’ research team, leveraging the company’s proprietary compound library. In preclinical studies, BLU-667 consistently demonstrated sub-nanomolar potency against the most common RET fusions, activating mutations and predicted resistance mutations. In addition, BLU-667 demonstrated markedly improved selectivity for RET compared to pharmacologically relevant kinases, including approximately 90-fold improved potency for RET versus VEGFR2. By suppressing primary and secondary mutants, BLU-667 has the potential to overcome and prevent the emergence of clinical resistance. Blueprint Medicines believes this approach will enable durable clinical responses across a diverse range of RET alterations, with a favorable safety profile.

Blueprint Medicines has an exclusive collaboration and license agreement with CStone Pharmaceuticals for the development and commercialization of BLU-667 and certain other drug candidates in Mainland China, Hong Kong, Macau and Taiwan. Blueprint Medicines retains development and commercial rights for BLU-667 in the rest of the world.

NantKwest to Present and Provide Update at Upcoming Jefferies 2019 Annual Healthcare Conference

On June 3, 2019 NantKwest (Nasdaq:NK), a leading clinical-stage, natural killer cell based therapeutics company, reported that Dr. Patrick Soon-Shiong and Company management will be participating in the Jefferies 2019 Annual Healthcare Conference being held in New York City from June 4th-7th, 2019 (Press release, NantKwest, JUN 3, 2019, https://ir.nantkwest.com/news-releases/news-release-details/nantkwest-present-and-provide-update-upcoming-jefferies-2019?field_nir_news_date_value[min]=2019 [SID1234536825]). The presentations and one-on-one discussions will feature a science/medicine review, along with a clinical update provided by Dr. Soon-Shiong and Company management.

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Conference Details:

Event: Jefferies 2019 Annual Healthcare Conference
Date/Time: Tuesday, June 4, 2019, Presentation at 8:30am ET
Location: New York, NY

Webcast Details:

A webcast of the Dr. Soon-Shiong’s presentation will available on the NantKwest website. A link to the live webcast can be accessed by visiting the Investors page of the Company’s website. In addition, a replay of the event will be posted on the website after the event and will be available for 30 days following the event.

For more information regarding NantKwest, please visit www.nantkwest.com.

Updated ASCO Guidelines Establish TAILORx-defined Cutoffs for Determining Chemotherapy Benefit with the Oncotype DX Breast Recurrence Score® Test in Node-negative Breast Cancer

On June 3, 2019 Genomic Health, Inc. (NASDAQ: GHDX) reported that its Oncotype DX Breast Recurrence Score test and the TAILORx results have been recommended to guide chemotherapy treatment use in patients with node-negative early-stage breast cancer by the American Society of Clinical Oncology (ASCO) (Free ASCO Whitepaper) in its 2019 Guidelines for Use of Biomarkers to Guide Decisions on Adjuvant Systemic Therapy (Press release, Genomic Health, JUN 3, 2019, View Source [SID1234536824]). Using the strong and highest level of evidence from TAILORx, the updated ASCO (Free ASCO Whitepaper) guidelines increase the proportion of women who can be effectively treated without chemotherapy based on the Oncotype DX results, highlighting the importance of testing all medically eligible patients as standard of care.

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"We are pleased that the ASCO (Free ASCO Whitepaper) guidelines have been updated to reflect the findings from the landmark TAILORx trial, the practice-changing results of which demonstrated that the Oncotype DX Recurrence Score result can be used to guide chemotherapy decision-making for all medically eligible women with the most common form of early-stage, invasive breast cancer," said Steven Shak, M.D., chief scientific officer, Genomic Health. "The TAILORx results have influenced positive treatment guideline recommendations from ASCO (Free ASCO Whitepaper) and other organizations around the world, elevating the Oncotype DX test to a new global standard of care."

TAILORx, sponsored by the National Cancer Institute (NCI) and conducted by the ECOG-ACRIN Cancer Research Group, involved 10,273 women across 1,100 trial sites in six participating countries. The study results, which were presented during the Plenary Session at the 2018 ASCO (Free ASCO Whitepaper) Annual Meeting last June and simultaneously published in The New England Journal of Medicine, demonstrated that the Oncotype DX Breast Recurrence Score test definitively identifies the vast majority of women with early-stage breast cancer who receive no benefit from chemotherapy, and the important minority of women for whom chemotherapy benefit can be life-saving. Patients with an Oncotype DX Breast Recurrence Score result of 25 or less – up to about 80 percent of patients – may be safely spared chemotherapy and its well-known side effects, while those with scores of 26 to 100 may receive a life-saving benefit from chemotherapy.

"The updated ASCO (Free ASCO Whitepaper) breast cancer treatment guidelines based on the TAILORx results will provide physicians with clarity and confidence that they are selecting the right treatment for each of their breast cancer patients," said Harold A. Burstein, M.D., Ph.D., medical oncologist at the Dana-Farber Cancer Institute. "Having guidelines that reflect the latest ground-breaking research is critical in ensuring that physicians incorporate standard-of-care technology, such as the Oncotype DX test, when making clinical decisions about treatment for their patients with breast cancer."

New TAILORx Data Analysis at 2019 ASCO (Free ASCO Whitepaper) Annual Meeting
New analysis of a secondary endpoint of the TAILORx trial will be presented in the "Breast Cancer—Local/Regional/Adjuvant" oral abstract session at the 2019 ASCO (Free ASCO Whitepaper) Annual Meeting. The presentation, titled "Impact of clinical risk category on prognosis and prediction of chemotherapy benefit in early breast cancer (EBC) by age and the 21-gene recurrence score (RS) in TAILORx," will take place on Monday, June 3. The abstract (#503) can be accessed here.

About Oncotype DX
The Oncotype DX portfolio of breast, colon and prostate cancer tests applies advanced genomic science to reveal the unique biology of a tumor in order to optimize cancer treatment decisions. The company’s flagship product, the Oncotype DX Breast Recurrence Score test, is the only test that has been shown to predict the likelihood of chemotherapy benefit as well as recurrence in invasive breast cancer. Additionally, the Oncotype DX Breast DCIS Score test predicts the likelihood of recurrence in a pre-invasive form of breast cancer called DCIS. In prostate cancer, the Oncotype DX Genomic Prostate Score test predicts disease aggressiveness and further clarifies the current and future risk of the cancer prior to treatment intervention, and the Oncotype DX AR-V7 Nucleus Detect test helps determine which patients with metastatic castration-resistant prostate cancer (mCRPC) are resistant to androgen receptor (AR)-targeted therapies. The Oncotype DX AR-V7 Nucleus Detect test is performed by Epic Sciences at its centralized, CLIA-certified laboratory in San Diego and offered exclusively by Genomic Health. With more than 1 million patients tested in more than 90 countries, the Oncotype DX tests have redefined personalized medicine by making genomics a critical part of cancer diagnosis and treatment. To learn more about Oncotype DX tests, visit www.OncotypeIQ.com, www.MyBreastCancerTreatment.org or www.MyProstateCancerTreatment.org.